This menu provides the list of EV miRNAs identified by high-throughput analyses.
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Search:
Dataset accession
miRBase accession
Mature name
Orthologous group
Superdomain:
All
Prokaryote
Eukaryote
Filter datasets:
- "Sample type" indicates the source from which EVs originated (e.g. B cell, serum).
- "Sample status" indicates the condition of the source from which EVs originated (e.g. Normal, miR-146a-treated, Patients of hernia).
By species
Homo sapiens
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By sample type
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Gastric cancer cells (SGC-7901)
Lung cancer cells (A549)
Mammary gland cells (MCF10A)
Mammary gland cells (NMuMG)
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Pancreatic cancer cells (AsPC1, BxPC3, Capan1, Capan2, MiaPaca2, Panc1, Pt45P1, A818, Colo357, HD3577)
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By sample status
24 hr culture
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Acute myeloid leukemia
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Normal_Exiqon
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Oral lichen planus patient
Pancreatic cancer patient_Exiqon
Transplant islets into mouse xenotransplantation-rejected
Type 1 diabetes
Maximum false positive rate (FPR):
Maximum true positive rate (TPR):
- FPR is the probability that an absent miRNA accidently have higher intensity than that miRNA.
- TPR is the percentile of miRNA among the present miRNAs.
Please see user manual in the contact us/help menu for detail.
Number of molecules in one page:
100
200
All
The downloaded CSV file is not exactly the same as the displayed table. Opening CSV file with Excel can impair its content.
Since there are multiple primers in the microarray, the same miRNA can have several FPR and TPR values.
Mature name
miRBase accession
FPR
TPR
Publication
Orthologous group
Identification count
All / Prokaryote / Eukaryote
(FPR<0.05,TPR<0.5)
hsa-miR-4454
MIMAT0018976
9.9e-7
3.3e-1
Plasma-derived exosome characterization reveals a distinct microRNA signature in long duration Type 1 diabetes.
Sci Rep. 2017 Jul 20;7(1):5998. doi: 10.1038/s41598-017-05787-y.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
1.2e-4
4.1e-1
Plasma-derived exosome characterization reveals a distinct microRNA signature in long duration Type 1 diabetes.
Sci Rep. 2017 Jul 20;7(1):5998. doi: 10.1038/s41598-017-05787-y.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
0.0e+0
1.1e-2
Tumor microenvironment interruption: a novel anti-cancer mechanism of Proton-pump inhibitor in gastric cancer by suppressing the release of microRNA-carrying exosomes.
Am J Cancer Res. 2017 Sep 1;7(9):1913-1925. eCollection 2017.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
0.0e+0
1.9e-3
Tumor microenvironment interruption: a novel anti-cancer mechanism of Proton-pump inhibitor in gastric cancer by suppressing the release of microRNA-carrying exosomes.
Am J Cancer Res. 2017 Sep 1;7(9):1913-1925. eCollection 2017.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
0.0e+0
4.1e-2
Tumor microenvironment interruption: a novel anti-cancer mechanism of Proton-pump inhibitor in gastric cancer by suppressing the release of microRNA-carrying exosomes.
Am J Cancer Res. 2017 Sep 1;7(9):1913-1925. eCollection 2017.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
0.0e+0
4.3e-3
Tumor microenvironment interruption: a novel anti-cancer mechanism of Proton-pump inhibitor in gastric cancer by suppressing the release of microRNA-carrying exosomes.
Am J Cancer Res. 2017 Sep 1;7(9):1913-1925. eCollection 2017.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
1.5e-3
1.5e-3
Circulating exosomes suppress the induction of regulatory T cells via let-7i in multiple sclerosis.
Nat Commun. 2018 Jan 2;9(1):17. doi: 10.1038/s41467-017-02406-2.
Not available
31
/
0
/
31
hsa-miR-4454
MIMAT0018976
6.1e-3
6.1e-3
Circulating exosomes suppress the induction of regulatory T cells via let-7i in multiple sclerosis.
Nat Commun. 2018 Jan 2;9(1):17. doi: 10.1038/s41467-017-02406-2.
Not available
31
/
0
/
31
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