This menu provides the list of EV proteins identified by high-throughput analyses.

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Superdomain: All Prokaryote Eukaryote
Filter datasets:
  - "Sample type" indicates the source from which EVs originated (e.g. B cell, serum).
  - "Sample status" indicates the condition of the source from which EVs originated (e.g. Normal, miR-146a-treated, Patients of hernia).
Number of molecules in one page:

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Protein name UniProt accession Publication Orthologous group Identification count
All / Prokaryote / Eukaryote
Splicing factor 3B subunit 4Q8QZY9Membrane microvesicles as mediators for melanoma-fibroblasts communication: Roles of the VCAM-1/VLA-4 axis and the ERK1/2 signal pathway.
Cancer Lett. 2015 Feb 4. pii: S0304-3835(15)00066-X. doi: 10.1016/j.canlet.2015.01.032.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9A comprehensive proteomics analysis reveals a secretory path- and status-dependent signature of exosomes released from tumor associated macrophages.
J Proteome Res. 2015 Aug 27.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9A comprehensive proteomics analysis reveals a secretory path- and status-dependent signature of exosomes released from tumor associated macrophages.
J Proteome Res. 2015 Aug 27.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Cells release subpopulations of exosomes with distinct molecular and biological properties.
Sci Rep. 2016 Mar 2;6:22519. doi: 10.1038/srep22519.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Cells release subpopulations of exosomes with distinct molecular and biological properties.
Sci Rep. 2016 Mar 2;6:22519. doi: 10.1038/srep22519.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Microvesicles provide a mechanism for intercellular communication by embryonic stem cells during embryo implantation.
Nat Commun. 2016 Jun 15;7:11958. doi: 10.1038/ncomms11958.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Secretome proteomics reveals candidate non-invasive biomarkers of BRCA1 deficiency in breast cancer.
Oncotarget. 2016 Aug 23. doi: 10.18632/oncotarget.11535.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Secretome proteomics reveals candidate non-invasive biomarkers of BRCA1 deficiency in breast cancer.
Oncotarget. 2016 Aug 23. doi: 10.18632/oncotarget.11535.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Thymic exosomes promote the final maturation of thymocytes.
Sci Rep. 2016 Nov 8;6:36479. doi: 10.1038/srep36479.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9The Hippo Pathway Kinases LATS1/2 Suppress Cancer Immunity.
Cell. 2016 Dec 1;167(6):1525-1539.e17. doi: 10.1016/j.cell.2016.11.005.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9The Hippo Pathway Kinases LATS1/2 Suppress Cancer Immunity.
Cell. 2016 Dec 1;167(6):1525-1539.e17. doi: 10.1016/j.cell.2016.11.005.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Proteomic profiling of exosomes secreted by breast cancer cells with varying metastatic potential.
Proteomics. 2017 Nov 8. doi: 10.1002/pmic.201600370.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Differential content of proteins, mRNAs, and miRNAs suggests that MDSC and their exosomes may mediate distinct immune suppressive functions.
J Proteome Res. 2017 Nov 15. doi: 10.1021/acs.jproteome.7b00646.
ENOG410XPT467 / 0 / 67
Splicing factor 3B subunit 4Q8QZY9Differential content of proteins, mRNAs, and miRNAs suggests that MDSC and their exosomes may mediate distinct immune suppressive functions.
J Proteome Res. 2017 Nov 15. doi: 10.1021/acs.jproteome.7b00646.
ENOG410XPT467 / 0 / 67
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